Strong acids
- Ex. sulfuric, nitric, hydrochloric, chromic
- Coagulate skin proteins → form a barrier that impedes further penetration
- Hydrofluoric acid causes liquefactive necrosis → penetration (even down to the bone) may last several days after injury
- R/ intralesional calcium gluconate
- If >1% BSA → risk of systemic toxicity
Strong alkalis
- Sodium, calcium, potassium hydroxides, wet cement, sodium and potassium cyanides
- Degradation of lipids → saponification of the remaining fatty acids → forms soaps which aid the penetration of alkalis deeper into the skin
- Damage more severe than acids
Organic and inorganic chemicals
- Ex dichromates, arsenates, phenolic compounds
- Nerve damage → anesthesia
- Vasoconstriction → necrosis
- Systemic absorption → shock and renal damage
Treatment
- Irrigation with large volumes of water
- Antidotes
- Calcium gluconate gel for hydrofluoric acid
- Debridement if risk of systemic toxicity